Genome

Pharmacogenomics: when your genes change how you respond to a drug

Star alleles, metabolizer status, and the ten FDA Black Box drug-gene alerts Haeckel surfaces. Concrete examples from CYP2D6 codeine and DPYD fluorouracil.

6 min read · updated Apr 19, 2026

Pharmacogenomics studies how variation in your genome changes the way your body absorbs, activates, breaks down, or responds to a drug. About one in five drugs sold in the United States carries a label warning that one or more genetic variants alter its safety or efficacy, and for ten of those the FDA has issued a Black Box warning that requires the prescriber to confirm the genotype before writing the prescription.

Star alleles, briefly

Most pharmacogenes have many catalogued variants, and the field uses a notation called star alleles to bundle each variant under a short identifier. CYP2D6*1 is the reference. CYP2D6*4 is a loss-of-function allele common in European populations. CYP2D6*10 is a reduced-function allele common in East Asian populations. A diplotype like CYP2D6*1/*4 names both copies you carry.

Each star allele carries an activity score that ranges from zero (loss of function) through 0.5 (reduced function) to 1 (normal) and beyond. Sum the activity scores across both copies and the result classifies you as a poor, intermediate, normal, rapid, or ultra-rapid metabolizer for that gene.

Concrete examples

CYP2D6 and codeine

Codeine itself is a prodrug. Your liver converts codeine into morphine using CYP2D6. A poor metabolizer experiences little to no analgesia. An ultra-rapid metabolizer can produce dangerously high morphine levels from a standard dose, which has led to fatal overdoses in children.

DPYD and fluorouracil

DPYD encodes the enzyme that breaks down 5-fluorouracil and capecitabine, two backbone chemotherapy drugs. About 1 in 30 people carries a DPYD variant that reduces enzyme function, and giving such a patient a standard chemotherapy dose can cause life-threatening toxicity. CPIC guidelines recommend a 50% dose reduction or switching agents.

HLA-B*57:01 and abacavir

About 5 to 8% of people of European ancestry carry HLA-B*57:01, a variant that is strongly associated with a severe and sometimes fatal hypersensitivity reaction to the HIV drug abacavir. The FDA mandates HLA-B*57:01 testing before prescription.

The ten Black Box alerts Haeckel surfaces

  • CYP2D6 with codeine, tramadol, and several antidepressants.
  • CYP2C19 with clopidogrel.
  • CYP2C9 with warfarin and phenytoin.
  • DPYD with fluorouracil and capecitabine.
  • HLA-B*57:01 with abacavir.
  • HLA-B*15:02 with carbamazepine in patients of East Asian ancestry.
  • TPMT with thiopurines.
  • NUDT15 with thiopurines.
  • UGT1A1 with irinotecan.
  • SLCO1B1 with simvastatin and other statins.
References
  • CPIC: Clinical Pharmacogenetics Implementation Consortium guidelines, cpicpgx.org.
  • PharmGKB: pharmgkb.org.
  • FDA Table of Pharmacogenomic Biomarkers in Drug Labeling, fda.gov.
Ask Mirror about this for your own genome

Are there any drugs I should be careful about based on my pharmacogenomic profile?